Your understanding of "established doctrine" is incorrect.
A handgun bullet, in a common combat caliber, can only be RELIED UPON to damage the tissues it comes into direct contact with.
It could cause more damage, but it depends on exact circumstances, which the shooter doesn't control and is therefore unreliable to depend upon.
Fackler identified a handful of factors in which the temporary cavity of a penetrating handgun bullet can cause more damage.
One of them is when the tissues involved are constrained, like intercostal tissues (the tissues between ribs). Intercostal tissues are short, connected to the ribs, and cannot stretch very far without tearing. Over the years I've encountered a lot of folks that post handgun hunting photos showing rib entrance wounds, on the exit side of the internal chest wall, that are larger in diameter than the bullet as "proof" that the temporary cavity of a handgun bullet causes greater permanent disruption than simply what the bullet comes into direct contact with.
The temporary cavity from a handgun bullet has the potential to damage (depending on exact circumstances):
- Soft tissues that do not tolerate being stretched
- Brain, liver, pancreas, kidney, spleen
- Elastic soft tissues that are tightly bound
- Unable to stretch far enough to absorb strain and tear/rupture
- e.g., intercostal tissues
- Size of elastic soft tissues being stretched
- e.g., individual muscles of the heart
- “Local anatomy”
Whereas resilient soft tissues can withstand the stretch of temporary cavity produced by handgun bullets with minimal damage:
- Lung, muscle, bowel, nerve, vessels
A penetrating handgun bullet has two different diameters: 1) its actual physical diameter, and 2) its "effective" diameter.
In elastic soft tissues, a handgun bullet's effective diameter is smaller than its physical diameter. This is because elastic soft tissues stretch and flow around the contours of the penetrating bullet. And as the bullet penetrates it slows, allowing elastic soft tissues more time to stretch and move out of the path of the oncoming bullet. As a result, the permanent cavity decreases in diameter the deeper the bullet penetrates. (The Winchester Black Talon-style bullet, with its sharp claws protruding outward, always has a larger effective diameter than other bullet designs because the claws cut resilient soft tissues that flow around the bullet.)
As for shooting a fast-advancing attacker, the tactic is to move laterally off the line of attack (side stepping) while shooting to stop him/her. This causes the attacker to run past the defender.
The aimpoint is the high upper torso, about armpit high on the attacker. This is where the specific vitals we're targeting are concentrated (heart, great vessels, and spine). Because both the defender is moving and the attacker is moving, this makes the attacker's head a challenging aimpoint to target, and bullets fired at the attacker are more likely to miss the head than the larger torso, and those errant bullets will continue flying until they hit something, hopefully not a person.
Yes, the heart and great vessels can be destroyed and the attacker can continue to attack. This is called post-fatal injury activity. John Lennon and Lee Harvey Oswald had their aortic arch destroyed. Lennon was able to walk into the lobby of his apartment building before he collapsed, whereas Oswald was quickly incapacitated.
On a frontal shot, the spinal column lies directly behind the heart and great vessels. Bullets that pass through these vital structures, or narrowly miss them can continue penetrating to hit the spine to disrupt the spinal cord (concussion of the spinal cord) and cause an advancing attacker to instantly collapse from flaccid paralysis.
The heart, great vessels, and spinal cord are all elastic soft tissues. These are the specific vital tissues we're trying to disrupt. Yes, there are other "vital" organs that are inelastic and can be damaged by the temporary cavity, but they do not hemorrhage in the same rate and quantity, especially if the attacker's sympathetic nervous system is activated, which decreases blood flow to these inelastic organs.
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