The hydrostatic shock theory?

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You are correct. But that is rupture of a vein caused by low-pressure, large-volume over-filling. The mechanism of a pressure wave is a momentary high pressure/low volume change; further, it is likely that the pressure wave would travel both within and along (outside) the vein, so there is no pressure differential to stretch the vein.

The importance of the vessels in the theory is that they allow the fluid pressure wave to reach the CNS, which is otherwise pretty well shielded by bone.
 
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Ok, but do you realize that BLOOD does not enter, EVER the CNS, and it causes bad thing, there are membranes etc. that form a 'blood brain barrier'

boys, we are talking jedi 'force' here
really lets stick to the stuff that matter instead of trying to develop some super round or debating intangibles

there may be a mechanism, but every RELIABLE study, and hunters account don't indicate some super CONSISTENT force, rather the same ol put a hole through something vital.
 
Ok, but do you realize that BLOOD does not enter, EVER the CNS
What an odd thing to say.

Of course blood enters the CNS--otherwise the brain and spinal cord would have no oxygen or nutrients, and could not exist. The blood that enters the CNS is (of course) contained within blood vessels.

What is meant by "blood-brain barrier" is that the CNS capillaries (unlike many capillaries elsewhere) do not leak large proteins (like albumin and immunoglobulins) into the surrounding tissue; small molecules (like oxygen and glucose) do cross the barrier.

But the blood-brain barrier would provide no protection from a pressure wave traveling along the CNS vessels.
 
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Another thing to keep in mind is that the pressures needed to disrupt nerve operation are VERY small.

The slight swelling around a damage site in MS creates enough pressure to disrupt nerve function over a few mm away.
this is one of the reasons that MS symptoms can appear to fade short term, but there can still be long term damage.

Nerves can also depolarize from physical blows.
just about everyone has bumped their 'funny bone.'
The slight impact causes the nerves to depolarize.
We perceive it as a 'shock' or any of the other hundreds of descriptions used.
We notice it because there are a lot of sensory nerves, but you may also notice that you cannot move certain portions of your hands after the blow.

Being knocked unconscious by a blow to the head is thought to have a similar physiology.
The nerves in the brain massively depolarize and stop working for a brief period.
Since these nerves are basic functions, you may not feel anything but will be unconscious.
As the nerves re-polarize and begin functioning you awake.

A blow that causes you to 'see stars' likely affected the visual paths in the brain, or the occipital lobes at the back of the head.
 
We seem to have "hydrostatic shock", "hydrodynamic shock", and "hydraulic shock" as terms...and I'm not sure that there's general agreement on what any of them are, and whether they are different. The Courtneys use "Ballistic Pressure Wave," and wiki uses hydrostatic shock.

Which is a part of the challenge. There is no general agreement on what is exactly being discussed and so there is no general agreed on terminology. So folks on both sides can argue past each other pretty easily. It's also possible for claims to gain some traction and have a certain shelf life that don't deserve it, due to misunderstanding. I'm referring here to the claim that a fella or animal shot in the hip, or foot or shoulder, with a high powered rifle round can be killed or knocked out due to a "pressure wave" that reaches the brain and bursts capillaries there, etc. This latter bit was a theory of Roy Weatherbys ,and some others, a number of decades back.

tipoc
 
As I understand it, a hydraulic system works by taking force applied to a master cylinder here and trasmitting it via displacement of hydraulic fluid from the master cylinder through lines to a slave cylinder over there.

That is correct, but don't confuse force with energy. Just because they are denoted with the same measurement (force is actually lb-ft/lb, poundals or other SAE and SI units) doesn't mean that they are synonymous. Kind of like ounce-both a mass and a volume unit, but 16 fluid ounces of petroleum oil does not weigh 1 pound, and a pound of mercury will not fill a pint container.

As well, a hydraulic system on a machine is closed and has a minimum of expandable chambers (namely the hoses, which don't expand much) so the pressure is transmitted with minimal loss. In a living organism, however, all of the tissues are compressable or elastic, especially the largest organ that contains it all-the skin. So when pressure is applied to a particular point on/in the body, the tissue and/or fluids that occupied that space simply move elswhere, and the pressure is largely dissipated over a much greater area than that being compressed.

As for the hydralic shockwave itself, we'll assume for the moment that the body is filled with fluid. The pressure created is obviously greatest at point of origin (bullet's meplat). Well, a bullet is very small, and the pressure will drop exponentially as the wave moves outward, and it also does so in a semi-spherical pattern. So that shockwave's energy and velocity is reduced exponentially as it moves further from the point of origin. Further reducing the actual effect of that shockwave is the fact that no soft tissue in the human body is truly rigid.

I certainly don't have hard figures on how much pressure a what velocity various organs and tissues can withstand before tearing/rupturing, but my own eyes and the experience of many others (experts included) seems to indicate that the hydraulic shock generated at normal handgun velocities is insufficient to cause the kind of peripheral wounding associated with high velocity rifle bullets where man-sized targets are concerned. Obviously, the aforementioned bunny simply lacks the overall mass to absorb the temporary cavitation caused by a handgun bullet-but as I mentioned in another thread, the affects of a rodent shot with a large caliber handgun are comparable to a human taking a howitzer round by scale. The temporary cavity at a given velocity is proportionate to the projectile size, so a 2" thick bunny getting hit with a .45 caliber bullet is like a normal sized person taking a 120mm shell.


So if your punch's force is transmitted by displacing a fluid-like material against the diaphragm...why is that not hydraulic?

I suppose we need to define fluid-like, because unless something is seriously wrong with you, your abdominal cavity is not filled with liquid. Are you considering internal organs to be fluid-like? Because I don't; They're a solid mass composed of cells. The cells may be primarily fluid in composition, but the membrane contains that fluid (a "sack", if you will). The only place where there is fluid within the torso (aside from blood in the vascular system) is between the membranes of the diaphragm (and boy does it hurt when an area geos dry [pleurisy]) But it's only a thin, lubricating amount.
 
Can you not think of it like this?
Take fire cracker and ignite it on the ground or empty can. Small damage.
Put that same fire cracker in a can full of water and you have a lot more damage and force given off.
 
Are you considering internal organs to be fluid-like? Because I don't; They're a solid mass composed of cells.
They're a fluid-like medium, in that if you strike them you get a shock wave to propogate. The standard ballistic "tissue stimulant" is a 10% gel, and we (along with everyone else) can argue whether a gel is "most like" a solid or a liquid. But there is little doublt that when you stirke a gel, you get a noticeable wave.
The only place where there is fluid within the torso (aside from blood in the vascular system) is between the membranes of the diaphragm (and boy does it hurt when an area geos dry [pleurisy])
Some false statements here.

Theres lots of fluid in the torso: fluid in the stomach, in the intestines--the intestines actually "float" in peritoneal fluid. As for the tissues themselves, they not only have fluid within the cells, but also "interstitial fluid" between the cells. And the cell membrane is also not solid.

Pleurisy is inflammation of the pleural space, and is typically accompanied by excess fluid there; it is not the pleura "going dry."

I'm not convinced the "pressure wave" theory is correct; but those who wish to convince people it's false seem to be in possession of a lot of anatomic misinformation.
 
They're a fluid-like medium, in that if you strike them you get a shock wave to propogate. The standard ballistic "tissue stimulant" is a 10% gel, and we (along with everyone else) can argue whether a gel is "most like" a solid or a liquid. But there is little doublt that when you stirke a gel, you get a noticeable wave.

I won't argue that you do cause a pressure wave when you strike soft tissue, but to that end, things that we consider true solids will also twist/contort with pressure wave, albeit not like soft tissue. However, I do not consider something that cannot flow to be a fluid/liquid; A kidney or lung does not sublime into a puddle when removed from the body and placed on a table. Internal organs and other soft tissues, similar to ballistic gelatin, exhibit properties between a liquid and a solid. They are soft and easily distorted, but more rigid than a substance like pudding or any true fluid.

Theres lots of fluid in the torso: fluid in the stomach, in the intestines--the intestines actually "float" in peritoneal fluid.

True. I suppose I should have been more specific in saying that the only part that is full of fluid is the Pleura. The stomach has only a small amount of digestive fluids that do not occupy it's entre space, nor is the peritonial "sack" full.

As for the tissues themselves, they not only have fluid within the cells, but also "interstitial fluid" between the cells. And the cell membrane is also not solid.

No argument. Just that those fluids are all separated by membranes or in between the cells; they do not constitute a significant reservior.

Pleurisy is inflammation of the pleural space, and is typically accompanied by excess fluid there; it is not the pleura "going dry."

Poor wording, it's just what I've always referred to the phenomenon as when it is that momentary occurance of the membranes sticking together that causes a sharp pain when breathing, but goes away relatively quickly. I'm not a doctor. lol. Just spent a fair amount of time in A&P books.
 
However, I do not consider something that cannot flow to be a fluid/liquid; A kidney or lung does not sublime into a puddle when removed from the body and placed on a table.

But they do flatten out and distort their shape vs. being supported in the body.

A water balloon would be a good example.
What percentage of the contents is water?
How does it behave when struck or force is applied?

Our tissue is overwhelmingly water, with cell membranes giving it some shape and form.

When it is struck, especially by fast moving objects like bullets, it behaves as a gel.

A material that is somewhere between a pure liquid and a solid.

Bones behave closer to a solid, but still contain marrow and pother cells that are NOT a solid (calcified).

Is cartilage a solid?
 
The importance of the vessels in the theory is that they allow the fluid pressure wave to reach the CNS, which is otherwise pretty well shielded by bone.

I'll point out again, that blood vessels are not empty hoses. Every few inches is a valve and if those valves are overcome it can be seen even from the outside of the body. If you know a current or former IV drug user, take a close look at their arms. Most of them will have concavities where their veins are, instead of the normal convex appearance of a healthy vein.

Of course, you wouldn't see this on an artery since they lie deeper in the body, but coroners would certainly note it on an autopsy.
 
The importance of the vessels in the theory is that they allow the fluid pressure wave to reach the CNS...
Vessels are not rigid pipes. They are elastic, which stretch and dampen the amplitude of the "blood pressure spike". They are also connected to other vessels, which disperse and dissipate the "blood pressure spike". In addition the lungs dampen the "blood pressure spike".
 
Every few inches is a valve

ONLY in VEINS.

Arteries do not have them (or need them).

The valves in veins allow for random muscle movements that can compress the veins to aid the return flow of the blood to the heart.

The valves allow one way flow, back towards the heart.

Just as a point, folks who have heart bypass surgery are kept on stool softeners for a few days.
The force of trying to defaecate could cause the new joints in the cardiac arteries to blow out until they have started to knit and heal.

that's right, pushing to poop could cause enough additional pressure at the heart to blow the connections.
 
But they do flatten out and distort their shape vs. being supported in the body.

A water balloon would be a good example.
What percentage of the contents is water?
How does it behave when struck or force is applied?

Our tissue is overwhelmingly water, with cell membranes giving it some shape and form.

When it is struck, especially by fast moving objects like bullets, it behaves as a gel.

A material that is somewhere between a pure liquid and a solid.

Bones behave closer to a solid, but still contain marrow and pother cells that are NOT a solid (calcified).

Is cartilage a solid?

An excellent perspective on the topic, the best that I've read in a long time. :)

In his book, "Bullet Penetration", MacPherson treats calibrated 10% ordnance gelatin as a "soft solid" with a high viscosity (~150 Poise) since it's able to sustain shear forces whereas fluids cannot. For the purposes of such discussion, it is a description that seems to "hold water" (I couldn't resist the pun, sorry :p) very well for how the material behaves under projectile impact forces.
 
When you shoot a handgun, how much energy is dumped in your palm? A similar amount of energy is dumped into the target (physics) and it isn't enough to make any difference. What counts is tissue destroyed along the path of the projectile, and that is not as simple as measuring kinetic energy.
What acts on your palm is momentum (Momentum = mass * Velocity.) Kinitic energy is calculated as Energy = mass * Velocity^2.
 
Vessels are not rigid pipes. They are elastic, which stretch and dampen the amplitude of the "blood pressure spike".
Very good point. I think the response would be along the lines of a "high-pass" filter: that a system that is flexible enough to totally absorb a low energy (slow) deformation may transmit a higher energy wave pretty well. So that a blood vessel that flexes in response to a heartbeat may act fairly rigid during the brief transmission of a ballistic pressure wave.

The high-pass aspect doesn't change the fact that the energy would attenuate with distance; but it would not dampen as quickly.
What acts on your palm is momentum
I think both kinetic energy and momentum would be imparted from the gun to my palm, and from there distributed elsewhere.
 
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The high-pass aspect doesn't change the fact that the energy would attenuate with distance; but it would not dampen as quickly.

And that one statement is what upsets and derails the ballistic pressure wave theory.

1) If a ballistic pressure wave originating in the chest is responsible for an "insult" of a certain magnitude being applied to the brain, then the same ballistic pressure wave originating closer to the head (such as the root of the neck) must produce a more overt "insult" to the brain. Where is the evidence for this? I personally saw a case where an adult male had most of his face removed by a contact wound from a .303 (ammunition unknown). His CT scan was clear (intracranially) and his GCS was normal for a person who is intubated (airway protection). Not one tiny little bleed was seen in the brain, yet the face was removed from the angles of the mandible all the way up to the nasion.

2) If a ballistic pressure wave originating in the chest is responsible for an "insult" of a certain magnitude being applied to the brain, then the same ballistic pressure wave must also apply the same "insult" to organs inferior to the chest, for example the kidneys. Where is the damage to the very fine vessels in the kidneys? Why is it we don't see haematuria (even microscopic) in patients who have sustained a GSW to the chest?

3) If a ballistic pressure wave originating in the chest is responsible for an "insult" of a certain magnitude being applied to the brain, then a greater ballistic pressure wave originating in the chest must produce a more overt "insult" to the brain. There should be overt and obvious findings of brain damage in individuals shot in the chest with rifle loads producing a greater ballistic pressure wave than handgun loads. Where are the reports (either from the ER or from the pathologist)?

This ballistic pressure wave theory has been touted by Michael Courtney on numerous boards over the years. He is the one who has altered/provided the most recent content in that Wiki also.
A few things to note (which I have posted on other boards also):

Over the years we have had many debates with Courtney, mainly on other boards.
When he first showed up on TacticalForums, everyone greeted him warmly and we were all receptive to his research.
It soon turned out that he was doing things backwards. He had arrived at a conclusion and was collecting disparate references to support it. What the pay-off would be if he got the theory accepted, I don't know for sure. It was one of two possibilities: either the ammunition or the treatment.
He could have wrapped this up years ago by submitting the heads of the shot deer for analysis by board certified veterinarians.
Every time I have made that suggestion he has come up with excuses...each one more ridiculous than the last.
First he said that he couldn't exclude head injuries on these deer from when they fell to the ground after being shot. I suggested a soft bait station but he said he isn't allowed to bait these. When asked about the veterinarian's input (or lack thereof) he claims financial limitations. He shies completely away from any kind of medical input whether it is expertise with animals or humans.
This is a guy with a PhD, doing a prospective research project. I recently completed a Master of Science degree in Health informatics and my final project was an expert system to do with the detection and enumeration of projectile fragments in humans from GSWs.
With these projects you have a proposal, a supervisor or advisor and a clear outline of what you are trying to do and what the acceptable measures of the outcomes will be. Even if he doesn't have to confirm to a university's work plan approval, his academic pedigree (which on paper is certainly higher than mine) should imbue his project with an element of academic integrity which needs an honest unbiased approach to the subject matter.
You don't on one hand claim that you cannot do the required testing on the deer because you will be hounded by PETA and then on the other hand announce that you have placed raccoons in buckets and fired into the water, resulting in the delayed death of at least one of those animals (again with no analysis by the required specialists to exclude pulmonary oedema or drowning as a cause of death).

I told him many moons back that I would accept his theory if he could show me one pair of deer that had been analysed post shooting (one by arrow, one by bullet), and there was definite evidence confirmed by an unbiased appropriate expert that the deer killed by bullet had neurological flags that the other one didn't. How hard is that to do, and is it really financially or academically taxing compared to the sum of all of his other efforts over the years related to this theory?

It wouldn't suit him to do it, because then his theory would be solidly rejected and he would get no foot in the door for whatever pay-off or commercial venture he envisages this BPW may unlock.

When I did my project, I provided a basic outline of what I was doing and also access to the associated online logic system I developed so that radiologists, trauma surgeons, AFTE, military and LEO persons could give me feedback and criticism on it. I used a database of 150 GSW patients where I had collected photographs of wounds, clothing, medical imaging and also operative and clinical notes and even photographs of recovered projectiles to do my study. I also had background statistics for the entire research period which encompassed an available sample of 542 patients. I collected this single-handeledly on site at a trauma unit in South Africa, with no funding and at the time with nothing more than a Diploma to my name.

Imagine how disappointed I was when I couldn't get the system to consistently match projectiles and trajectories in circumstances where a person may have more than three skin breaches.
Instead of trying to peddle this system (I have had a lot of interest in this work) I have had to back off because I can see that this isn't going to work. I may have to try something different, such as doing another project where I can get ethical clearance to change the way in which routine X-ray imaging is done for a prospective sample of GSW patients.
That is the correct thing to do.

Disparate references, meandering around available resources which could prove or refute his theory in short order, and dare I say it a certain lack of academic integrity and ethics on the part of Courtney have led me to take an adversarial stance against him.

Whilst I have seen traumatic effects one or two inches from a bullet's terminal trajectory in real patients in a variety of tissues, there is nothing in the totality of the evidence available to support Courtney's BPW theory and I for one don't buy it.
He published it in a journal, yes, but that doesn't mean it is sound. You can check the articles by Wakefield and the MMR scandal here in the UK if you want an example of less than forthright academic publishing.

Courtney scoured all the literature and all he could find was one gunshot face from the 40s, reported in the 90s IIRC, with a supposed link between that injury and a subsequent finding of epilepsy in that patient. I posted here on GT an exact reconstruction of the trajectory of that bullet based on the author's own information in the article and guess what I found in the path of that bullet: vertebral arteries. It is disengengious for Courtney to even cite that reference because there was no computed tomography in the 40s when the injury was acutely treated, and there was no digital subtraction angiography either. It is further compounded by his apparent disdain for other peoples' offering of Vietnam-era data where he claims the imaging and diagnostic techniques were not sophisticated enough to detect BPW effects. When it suits him, the imaging and clinical data is enough, when it doesn't suit him not only does he claim we aren't doing enough to detect this, but he offers no prospective study of his own to confirm it.

And here is the recon:

MIP.jpg

This is my summary of the state of play at present:

At this point the BPW theory is academic, Courtney has moved on. Whatever pay-off he envisaged getting once he got the theory accepted in the domain is clearly unattainable now. The institutions/agencies that would have been necessary to have buy-in obviously didn't go for it and Courtney has moved on to other things.

We all agree in principle to use a service calibre weapon with reputable expanding ammunition with the potential to penetrate to the vitals. If you believe that ammuniton X satisfies that and also provides a certain percentage/probability of causing incapacitation by remote neural/vascular damage, then go for it and get that. Everything else is just wasted bleating.

If you want to see the whole gory thread, it is on GT:
http://glocktalk.com/forums/showthread.php?t=1346952&page=11
 
Are there no trauma surgeon/gun enthusiasts on here that can put this to rest once and for all?

I've read stuff from a morgue worker and he said that rifle fire was almost always lethal and almost always killed in one round. Now correct me if I'm wrong, but a 7.62 is smaller diameter than a 9mm, but both flavors in FMJ will over penetrate.

Then the 9mm should be better than the 7.62. But it isn't.

He said that rifle victims were rare, but were almost always instantly lethal. He said that a 7mmMag victim had liquified organs. Massive trauma. Most rifle and shotgun wounds he saw were head shots, suicides. But the few that came through with COM shots were nasty.

He also noted that 9mm victims came in with several shots. Usually .40 and .45 were one or two shots. Obviously whoever was firing didn't feel the 9mm was putting them down fast enough.

So it would seem for pistols, that bigger is better. For rifles, faster is better. I think this arguement has become a cycle, I've been interested in this kind of thing since I first started shooting when I was 8yo. I've read about it until the internet came along, then discussed it. We're still where we were 20 or 30 or more years ago.

That really is the bottom line. Until we get a trauma surgeon on here to set us straight, it'll just have to be a little mysterious yet.
 
I can't offer anything but a second-hand anecdotal account, but it sounds like that's what you're looking for: when the wife was in med school, she had one week where she had one patient who had been shot with a 12ga shotgun, and another that had been shot with an "AK47" (so figure an SKS or something.)

Her assessment at the time: she'd rather take the close-range buckshot then the rifle round. In the end the rifle victim had to keep going back through surgery to have additional tissue removed, with the end result of it being a more devastating wound.

Anecdotal, and completely dependent on my memory (I asked her and the events in question have all blended together with the rest of the crap she saw in training), but I was convinced.
 
Appreciate the perspective, Odd Job. Good stuff to chew on. Most of the response would I think again center on concussion: that loss of consciousness can at times be observed (with direct blunt trauma to the skull) even without cerebral contusion or other clear signs of injury (other than changes in the concentrations of some neurotransmitters and some inflammation mediators).

It remains the case that, even if pressure-wave-induced unconsciousness occurs, it seems to be an undependable phenomenon.

(Oh--and I always thought Odd Job was the best Bond henchman. Way better than Jaws. :))
 
Can you not think of it like this?
Take fire cracker and ignite it on the ground or empty can. Small damage.
Put that same fire cracker in a can full of water and you have a lot more damage and force given off.
moot point. This is not a description of hydrostatic shock. The gases caused by the rapid conflagration of the firecracker simply displace the water. Since water cannot be compressed, it has to move out of the way. This is why it ruptures the can. As for more force given off, that's simply impossible. The same type of firecracker will give off approximately the same force. It draws no other strength from the water it's in.
 
.I think both kinetic energy and momentum would be imparted from the gun to my palm, and from there distributed elsewhere.

If that were true, your palm would suffer as much damage as the target.

The bullet and the gun have about the same momentum. The momentum of the gun will be a little higher because of the momentum of the powder gasses that act primarily on the gun.

K.E. is a totally different situation. Since it's based on velocity squared, the bullet has much more energy than the gun. In the earlier post with the .357 mag example the velocity and K.E. numbers are:

2 lb .357 mag at 17.25 fps = 9 ft-lbs
158 gr bullet at 1250 fps = 548 ft-lbs



The K.E. of the gun is also distributed over the surface of your hand in contact with the gun. The K.E. of the bullet is distributed over a much smaller area.
 
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