First, where do those wound channel illustrations come from and what are they based on? Clearly, they are not photographs of damaged tissue. You can see in most of them little diagrams of a tumbling but intact projectile or a fragmenting projectile. In the case of the intact projectile, what appears to be a representation of the primary crush channel is relatively small whereas when the bullet fragments it is quite large. So what is that envelope around the primary crush channel delineated by the second line and what is it based on? I suspect it is based on the so-called secondary wound channel seen in ballistic gel. You say it is "an area of destruction caused by a very large cavitation that exceeds the stretch capacity of the tissue." And what I am telling you is that it does not exist, or frequently doesn't, at least not as an area of tissue destruction that is visible at surgery. It is never seen with the common handgun self-defense cartridges. And even intermediate power rifle bullets traversing soft tissue that do not yaw, tumble, or fragment very commonly do not have any area of tissue destruction caused by a very large area of cavitation. At most, there might be a little devitalized muscle tissue immediately adjacent to the primary crush channel. I have been there and seen this with my own eyes. If there was significant tissue destruction due to a large area of cavitation, then the conservative treatment of these rifle wounds with minimal excision of entry and exit sites, and irrigation would never work. But it frequently does work. Here is a short article on the management of high velocity gunshot wounds of the extremities:
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4596205/
And here is a paragraph taken from that article:
"An injury with small entrance and exit wounds, no neurovascular compromise, and no evidence of fracture or bullet fragmentation on radiographs can be regarded as a simple through-and-through wound. It is likely in these injuries that there is a small amount of necrotic tissue in the permanent tract only and they can safely be managed minimally with irrigation, dressings, and prophylactic oral antibiotics."
And speaking of bad examples, I don't think your comparison of what happens when someone gets the wind knocked out of them by a punch to the gut has any bearing on what happens when someone gets shot in the abdomen. Although only an elite boxer can deliver close to the kinetic energy of even most handgun projectiles with their fist, arm, and body throwing a punch, that energy is delivered to a broad area of the skin of the abdominal wall and does not penetrate the skin. It therefore causes a large degree of displacement of the abdominal wall and a greater transient increase in intra-bdominal pressure than even a high velocity projectile striking and penetrating the abdominal wall. It is this large increase in intra-abdominal pressure that pushes up on the diaphragm emptying the lungs of air and sometimes temporarily interfering with diaphragmatic function. The increase in intra-abdominal pressure can also cause an immediate cessation of blood return from the abdomen and lower extremities via the vena cava.
Lets see if we can settle on a few points of agreement. I hope you would agree with me that pain and fear can be temporarily incapacitating. It seems that you found this out when you had a surgical drain removed, and having removed many, many drains I can tell you that it did not result in any cavitation, as you already know. Even surprise can be incapacitating to immediate action. We know from a number of well-publicized events that not everyone who gets shot feels pain, fear, or even surprise. But I think it is reasonable to propose that many do. And if they do, these can all be temporarily incapacitating factors apart from the purely psychological effects of getting shot.
As for whether or not rapid tissue displacement that does not cause permanent tissue injury (by whatever mechanism we wish to invoke) can have a temporary incapacitating effect, you can't provide any incontrovertible evidence for its existence (at least not any I would consider incontrovertible) but you seem to feel strongly that such an effect does exist. And I certainly can't provide any evidence that it does not exist.
You seem to think that I believe strongly that such a mechanism does not exist. That is actually not true. I have taken into account the accounts of various hunters and a few anecdotal stories of humans who have been shot with various high velocity handgun cartridges. I am more of an agnostic when it comes to this issue having simply seen no solid evidence for its existence.
The problem with evaluating the outcome of individual gun fights involving humans at least, is we know there is such a vast difference in the terminal ballistic behavior of cartridges from case to case, and a huge variation in how individuals respond to being shot. We all know about the case of Michael Platt. If you go to youtube and search for "The murder of Trooper Mark Coates" you will see a dashcam video of a South Carolina State Trooper who made a traffic stop of an individual who attacked him with a 22 LR handgun. Trooper Coates hit the perp 5 times center mass with 357 Magnum silvertips at point blank range without any apparent effect. Coates was killed by a single 22 LR shot that penetrated his ascending aorta, and the video shows that he was on the ground within 30 seconds. In another documented instance, a perp was shot 6 times in the chest with 45 Long Colt at point blank range, shot between the shoulder blades 5 times with 38 Special +P at contact range, then shot twice more with .44 Magnum rounds at a range of 15 feet and rapidly closing. He was a small individual and none of these 13 center mass hits had any apparent effect. He was shot twice more in the thigh and knee and it was the 15th shot to the knee that took him out of the fight. He lived for 10 days. It is anomalous outcomes like this that make it difficult for me to know how much importance to attach to anecdotal accounts of shootings.